Doctors are currently treating COVID incorrectly according to recent research. COVID patients actually suffer from "hypersensitivity pneumonitis" due to histamine release by neutrophils (see sources, below - this is a new discovery that researchers made) which are attacking infected cells in their lungs.
SOURCES: Please note that sources directly concerning COVID are being put together as they come in and there is a chance they could be inaccurate simply based on the constantly evolving nature of COVID infections and their treatment.
Neutrophil-to-lymphocyte ratio and lymphocyte-to-C-reactive protein ratio in patients with severe coronavirus disease 2019 (COVID-19): A meta-analysis.
Dysregulation of immune response in patients with COVID-19 in Wuhan, China
Severe cases tend to have lower lymphocytes counts, higher leukocytes counts and neutrophil-lymphocyte-ratio (NLR), as well as lower percentages of monocytes, eosinophils, and basophils.
Univariate analysis showed patients with ocular symptoms were more likely to have higher white blood cell and neutrophil counts, as well as high levels of procalcitonin, C-reactive protein and lactate dehydrogenase, compared with patients without ocular symptoms.
Jackson Hall
Bump for interest
Daniel Sanders
Distinct severe acute respiratory syndrome coronavirus-induced acute lung injury pathways in two different nonhuman primate species.
Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS), caused by influenza A virus H5N1 and severe acute respiratory syndrome coronavirus (SARS-CoV), supposedly depend on activation of the oxidative-stress machinery that is coupled with innate immunity, resulting in a strong proinflammatory host response. Inflammatory cytokines, such as interleukin 1β (IL-1β), IL-8, and IL-6, play a major role in mediating and amplifying ALI/ARDS by stimulating chemotaxis and activation of neutrophils.
Julian Evans
Effects of ibuprofen on neutrophil function and acute lung injury in canine endotoxin shock.
The role of the polymorphonuclear leukocyte in the development of acute lung injury has been the subject of much controversy. Experimental lung injury is associated with peripheral leukopenia and the intrapulmonary sequestration of leukocytes. We have previously shown that ibuprofen, a nonsteroidal anti-inflammatory drug, can improve the hemodynamic alterations of canine endotoxin shock. Ibuprofen has also been found to decrease leukocyte adherence. We investigated the dose response of ibuprofen on the increased neutrophil adherence and the extent of lung injury associated with canine endotoxin shock. Single doses of ibuprofen (1, 5, 10, and 20 mg/kg iv) were administered 15 min after Escherichia coli endotoxin. Endotoxemia resulted in leukopenia and an increased neutrophil adherence in both aortic and pulmonary artery blood. Endotoxin-treated animals exhibited increased neutrophils in the bronchoalveolar lavage fluid, a marker of lung injury. The 20-mg/kg ibuprofen dose decreased aortic granulocyte adherence at 30 min, while all ibuprofen doses decreased the aortic adherence at 120 min. The increased pulmonary artery neutrophil adherence was not affected by ibuprofen. Histologically, lung injury was manifested by intravascular leukostasis. Ibuprofen treatment did not affect the histologic or morphometric extent of the lung injury. The leukopenia and increased neutrophil adherence occur rapidly after endotoxemia and are associated with subsequent intravascular sequestration of leukocytes. Agents designed to prevent lung injury must either be given before the insult or be able to block the effects of the toxic products released by the activated granulocytes.
Lucas Moore
Cimetidine does not impair lung host defense in experimental pneumococcal pneumonia.
Effects of antihistamines on the lung vascular response to histamine in unanesthetized sheep. Diphenhydramine prevention of pulmonary edema and increased permeability.
To see whether antihistamines could prevent and reverse histamine-induced pulmonary edema and increased lung vascular permeability, we compared the effects of a 4-h intravenous infusion of 4 mug/kg per min histamine phosphate on pulmonary hemodynamics, lung lymph flow, lymph and plasma protein content, arterial blood gases, hematocrit, and lung water with the effects of an identical histamine infusion given during an infusion of diphenhydramine or metiamide on the same variables in unanesthetized sheep.
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Anthony Carter
Histamine caused lymph flow to increase from 6.0+/-0.5 to 27.0+/-5.5 (SEM) ml/h (P less than 0.05), lymph; plasma globulin concentration ratio to increase from 0.62+/-0.01 to 0.67+/-0.02 (P less than 0.05), left atrial pressure to fall from 1+/-1 to -3+/-1 cm H2O (P less than 0.05), and lung lymph clearance of eight protein fractions ranging from 36 to 96 A molecular radius to increase significantly. Histamine also caused increases in lung water, pulmonary vascular resistance, arterial PCO2, pH, and hematocrit, and decreases in cardiac output and arterial PO2. Diphenhydramine (3 mg/kg before histamine followed by 1.5 mg/kg per h intravenous infusion) completely prevented the histamine effect on hematocrit, lung lymph flow, lymph protein clearance, and lung water content, and reduced histamine effects on arterial blood gases and pH. 6 mg/kg diphenhydramine given at the peak histamine response caused lymph flow and lymph: plasma protein concentration ratios to fall. Metiamide (10 mg/kg per h) did not affect the histamine lymph response. We conclude that diphenhydramine can prevent histamine-induced pulmonary edema and can prevent and reverse increased lung vascular permeability caused by histamine, and that histamine effects on lung vascular permeability are H1 actions.
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Dominic Gonzalez
You'll still get sick like a dog. But at least you won't end up in the ICU. This protocal works.
Any questions? Feel free to ask.
Elijah Howard
Save this information. You might need it in the near future. Most of the drugs mentioned are OTC drugs. I hope that some of you are paying attention to this post and saving the info. It might save your life.
Kevin Stewart
Sooooo... take benadryl?
Adrian Sanders
This treatment should keep you from getting any worse and getting in the hospital.
Aaron Rivera
Translate this to English. I'm interested
Asher Ramirez
Benadryl (no cough suppressant – Original Formula) Diphenhydramine
Tagamet HB (H2 Antagonist) Cimetidine
Ibuprofen
In that order. Please watch the video. The key is taking these meds before your situation deteriorates. The reason people end up in the ICU is because of the inflammatory response from your body.
Cameron Jones
Yeah add a tl;dr on what cure to take for retards like me cause you posted so much shit at once that it looks like some pre made shill tactic to spread disinfo.
Jose Anderson
Before i try to read this science textbook you've copy pasted do you mind just saying in lay english what the protocols are so i can decide if this is plausible or schizo before i invest an hour deciphering it?
Thanks.
Logan Rodriguez
Tocilizumab is currently working the best in hospitals, and it's an IL-6 blocker to stop the cytokine storm.
Ethan Robinson
It's not. I posted very relevant information to support my point and my thesis. Anyone in my field , will quickly be able to understand what I am saying, or refute this information. I'll make a TLDR statement.
Josiah Diaz
This study literally says H2 antagonism didn't do anything compared to control. If anything the effect is limited to H1.
Noah Roberts
I have an albuterol nebulizer, I was planning on using.
Robert Bennett
The minute that you start getting sick, sore throat, start with these two medications:
Benadryl and Tagamet HB
DO NOT TAKE IBUPROFEN AT THE BEGINNING. BECAUSE IT IS INHIBITING ANTIBODIES SYNTHESIS. This is not what you want to happen in the first stage of infection. You need your immune system to be fully operational.
8 days later, the virus is not longer replicating in the average body's patient. Serious side effects usually start at this stage. Your immune system is drained. Expectthe following symptoms: trouble breathing, general exhaustion. This is when you want to start taking Ibuprofen. Why?
Take is as prescribed. This is ALL on you. If you die from it, that's not my problem. Why people are dying is not from because the virsu destroys your lung tissues. Your own body response is.
The allergic reaction from your body is making your lungs swell, making you unable to breath.
That's the best that I can do to simplify this information for you. I am not a doctor, so I am not responsible for you making decision that could affect your life and the outcome if you catch this disease.
Aaron Butler
Everything I've read has said that ibuprofen worsens the inflammation, and you should take acetaminophen. I had it for a week, I took acetaminophen, dextromethorphan, guaifenisin, and lots of vitamin c and zinc and turmeric, while drinking a lot of water, chamomile tea, and homemade chicken broth. There were a couple of times I thought I was going to die, but I refused to go to the hospital and just stuck my face near the tea pot and suck up all the steam until I could breathe. I'm certain that if I went on a ventilator, I would have died, lots of people are dying from the ventilators. Like you said, the virus basically makes your immune system attack and inflame your lungs, and it's the inflammation that's deadly, so you need to reduce the inflammation with acetaminophen, turmeric, and chicken broth, and reduce coughing with cough suppressants to reduce further inflammation damage, vitamin c and zinc to bolster immune system against anything that might want to take advantage of your inflamed lungs and start an infection like pneumonia. A month has passed since my symptoms went away, and I feel fantastic. Broth is easy to make and has always been my go to cold and flu remedy, boil the shit out of a chicken carcass and as many bones as you can throw in, make sure to leave a lot of cartilage, and also have various vegetable scraps boiling with it, and some salt to taste, and you have something you can drink by itself or a starter for all kinds of soups. I'll make a bunch and freeze it, thawing and heating when I need it.
Cameron Flores
>zinc
Zinc and Quercetin works as well. Quercetin is a chelator, allowing the Zinc to enter the cells. This is the same process witnessed with Hydroxychloroquine.
Chase Parker
A made-in-Canada solution to the coronavirus outbreak?
Quercetin isn’t the only possible treatment for COVID-19; Nature reported that 80 clinical trials on potential treatments are underway in China. But it remains one of the biggest potential leaps in finding a treatment for the deadly coronavirus strain; if it works, it could save thousands of lives.
Evan Reyes
I included this study to show that it's wrong.
Alexander Edwards
Is quercetin over the counter? I won't go to a doctor unless my arm is falling off. I'm sure this virus will be endemic, so it helps to have a plan for the next time.
Good. But not at the first stage of infection. I have already answered that question. Read the thread.
The minute that you start getting sick, sore throat, start with these two medications:
Benadryl and Tagamet HB
DO NOT TAKE IBUPROFEN AT THE BEGINNING. BECAUSE IT IS INHIBITING ANTIBODIES SYNTHESIS. This is not what you want to happen in the first stage of infection. You need your immune system to be fully operational.
8 days later, the virus is not longer replicating in the average body's patient. Serious side effects usually start at this stage. Your immune system is drained. Expectthe following symptoms: trouble breathing, general exhaustion. This is when you want to start taking Ibuprofen. Why?
Take is as prescribed. This is ALL on you. If you die from it, that's not my problem. Why people are dying is not from because the virsu destroys your lung tissues. Your own body response is.
The allergic reaction from your body is making your lungs swell, making you unable to breath.
That's the best that I can do to simplify this information for you. I am not a doctor, so I am not responsible for you making decision that could affect your life and the outcome if you catch this disease.
Everyone knew NYC was faking it at first to make trump look bad. Politicians are piggies. You’re a turd for trying to trick fatties you meme fart
Gavin Turner
With that flag LMFAO
Blake Sanders
>Covid 19 causes pneumonia >Nigger grandma with covid dies from pneumonia >Retarded nigger grandkid who doesn't understand how the virus works says corona-chan a gud gurl she dindu nuffin >Covid mostly kills people with other health problems >This means that covid had absolutely nothing to do with their deaths! You fucking retards who constantly deny anything is happening, make it worse for the rest of us by distracting from what the glowies are doing to take advantage of the situation. It's a real disease and it's really killing people, you retard.
Grayson Torres
COVID-19 patients aren't suffering from viral pneumonia. They are suffering from something called: Hypersensitivity pneumonitis
Spastic shitting up this thread: STFU. The government reaction to this can be overblown and opportunistic AND the virus can actually be deadly for a small portion of the population. Both things can be simultaneously true. Any genuine info on how to treat it isn't worthless. I don't know if OP is a fag or not, but you sure are.
Sebastian Brooks
He's spamming other threads with this shit. Clearest case of controlled opposition glowie shilling I've ever seen.
The reason why one of the most prevalent symptom is a dry cough is because unlike the flu, or TB, the lungs aren't filled with secretions. The reason why people are unable to breath is due to lung inflammation.
