Spatial modeling suggests COVID attacks hemoglobin:
chemrxiv.org
NYC Doctor (among others) say symptoms bizarre and do not resemble ARDS/pneumonia, but instead altitude sickness/hypoxia:
youtube.com
Artificial intelligence finds HIGH HEMOGLOBIN (and ALT, myalgia) predictive of severe outcomes:
healthitanalytics.com
Other urls found in this thread:
theatlantic.com
cdc.gov
ncbi.nlm.nih.gov
archive.is
ncbi.nlm.nih.gov
sciencedirect.com
youtube.com
youtube.com
youtube.com
healthitanalytics.com
spectator.co.uk
archive.4plebs.org
twitter.com
Makes sense if hydroxy chloroquine is actually working. Since it treats malaria, also a bloodborne disease.
We should be able to test this pretty readily with a simple transfusion. Hoping someone tries it. This could be something.
If it attacks hobgoblins will it keep the booger man away?
Keep taking your antipsychotics
You son of a bitch
>And guess what he found? Men have an average of 13.6 to 17.5 grams of hemoglobin per decalliter in their blood. Women have 12.0 to 15.5 g/dl.
The ratio? .88 to .89.
And while maximum oxygen consumption statistics are harder to measure and harder to come by, if you compare them for four accomplished long distance runners of each gender, they average to 72.7 for women and 82.1 for men. 72.7 is about 89 percent of 82.1.
Also this. Men have a higher % of hemoglobin in the blood as compared to women. May explain the higher fatality among men vs women.
theatlantic.com
>The Atlantic
I know.
My hemoglobin level was 14.1 last time I had a blood test.
Will I die of coronavirus?
Higher baseline %/decaliter* that is.
Aint no one doing anything based off an ai prediction. Just not going to happen.
Plaquenil interferes with membrane interactions in malarial infections which prevents the parasite from entering the cell.
While viral toxins may affect hgb, it would be odd for it to directly infect rbcs as there is no nucleus and rbc lack the capacity and protiens for replication, unless there is an infection of erythropoeitic stem cells, and that would be difficult to prove and would require bone marrow, which likely has not occurred.
Treatment protocols have been continuously adjusted by ICU physicians and be it ards/pna or pulmonary edema the treatment and managment of respiration and oxygen delivery has been adequate. To call this and treat it as altitude sickness would kill people as it clearly from a physics perspective is not the same thing as respiratory failure produced by high altitude.
>started shaking uncontrollably
Bloo blah blah hehehehuhahahaa me likey conspiracy XD upboated
Someone on here posted some bullshit about epogen being a possible treatment/cure the other day. Any anons think this is valid?
I thought of all this first, ages ago.
But he's right. The hypoxia seems to come first and the respiratory symptoms follow.
HCQ + Zinc would be really effective if it were attacking hemoglobin. HCQ alone would not. Ventilation would make it worse. Anyone with Italian, Spanish, Middle Eastern or African background with high chance of having sickle cells would be more at risk of severe symptoms and more likely to die - especially if old.
It turns out that's what we're seeing. Blacks, Hispanics, and Arab Muslims are disproportionately dying in America, France and the UK. Spain, Italy, Iran are all getting hit hard - mostly among those populations.
It seems like your blood is going to predict who will get the most sick, and age + other underlying conditions (whether you know about them or not) will predict who dies.
If everyone took HCQ + Zinc at the first sign of symptoms, death rate could drop to almost 0.
Do you have anemia, caused by AIDs? If not, then I wouldn't listen to anything posted on this site as something you should do.
How would the AZ pack stack with the Hydroxy/zinc? Would it even be needed?
The azithromycin was being used as an anti-inflammatory if I remember right, but it's an antibiotic. If it can be avoided, I would.
AIRBORN AIDS
Clinical Virology
Jeffrey K. Actor PhD, in Elsevier's Integrated Review Immunology and Microbiology (Second Edition), 2012
Coronavirus
Coronaviridae are best known as the second most frequent cause of the common cold. Although coronaviruses are known to cause disease in birds and pigs, recent studies indicate that the agent responsible for the severe acute respiratory syndrome (SARS) belongs to this genus. SARS is a serious, life-threatening viral infection that is thought to have mutated to a human transmissible form. The mortality rate of SARS is high compared with other common respiratory viral infections, with up to 10% of fatalities due to respiratory failure after symptoms of hypoxia, cough, and dyspnea (labored breathing).
>sciencedirect.com
I know its hard to understand, but the lungs are how your body get oxygen.
Now, listen.
Would a threatment using erythropoietin be safe?
>What is oxygen saturation
If your oxygen saturation falls below 80% you start to see organ function compromised. Oxygen saturation and hemoglobin are directly linked.
I heard the zpack is hard on the heart tho
airborne aids, 5g assist oxygen depletion
Huh, no wonder blood type A Positives are more susceptible to this.
what if you have low hemoglobin? im cirrently due for a blood transfusion because i bleed so much out my ass all the time with my bowel disease that i need blood every year or so, but i havent went to hematology or emergency room because of coronachan
Thank you user. We'll quote you on this.
So basically it's spread by insects also and can't die in summer
Reanimate this thread pl0x.
bump
Blueblood is immune, get rekt peasants and mutts
My Hemoglobin was 18.3 at my last physical. I’m proper fucked.
If so, Fuck Florida!!!
Just like malaria- wish there was a drug for that
covid is whatever you want it to be
>anemia, caused by AIDs
YES
Latest Study from Deutschland's epicentre.
Corona Chan IS attacking the blood.
Mine is 15-16
Good bye
the new home appliance
COVID doesn't attack anything you fucking glownigger. The only thing that needs to be attacked is your face. With a fucking bullet. In minecraft.
Very simple and efficient theory. BTFOs all the cretins who treat it with ventilation.
bump
There is nothing in his video about the virus binding to hemoglobin. Just some hopium about immunity for a corona viridae as if that is possible.
Bump
Several paper reviewed on drug models the behavior of chloroquine and faviparavir as well, which appear to bind to the non-structural viral proteins that hijack the heme groups, thus inhibiting them from knocking out the iron and wrecking the O2-carrying ability of the red blood cells.This also explains the observation made by various ER docs that patients tend to have elevated ferritin: ferritin is used to store excess iron released from the RBC damage. If a lot of iron is knocked out of heme groups and floating around, the body produces more ferritin.In humans, it acts as a buffer against iron deficiency and iron overload. This overlaod happens hyperacutely in COVID19. Ferritin is found in most tissues as a cytosolic protein, but small amounts are secreted into the serum where it functions as an iron carrier. Plasma ferritin is also an indirect marker of the total amount of iron stored in the body; hence, serum ferritin is used as a diagnostic test for iron-deficiency anemia. This anemia can happen acutely or chronically.
If this mechanism of damage is true, this implies we need to think a lot differently than we are now:1. Starting drug treatment while symptoms are mild keeps virus from hijacking too much of the RBC and cytochrome C oxidase, or catalase, enabling a still-healthy body to mount an immune response. Plasma therapies should be done before RBC transfusions.This explains why early drug treatment (first week of symptoms) is often successful. It stabilizes iron metabolism and stabilzes CO2/O2 delivery early on and maintains mitochondrial function via cytochrome C oxidase. This preserves ATP function. Red light therapy would augment this affect. Nitric oxide mimicry might save cytochrome c oxidase. This might be why UV light helps limit COVID 19 cases. UV light increases the production of nitric oxide and this shuts down the use of cytochrome c oxidase.
2. Drug treatment and intubation once patient is critical these option will rarely work because tissues/organs are already damaged by viral destruction of the beta chain of hemoglobin; therefore, blood can't carry O2, and the body is too weak to produce new red blood cells able to carry iron (and thus oxygen/CO2) even if drugs inhibit more hijacking.3. Thus: start severe patients on drug treatment upon hospital intake to suppress further hijacking of blood by the virus, then give them a blood transfusion of new red blood cells immediately that are unhijacked.4. If heme/hemoglobin is involved, a higher hemoglobin count may be protective to the disease process. This may explain why nicotine helps. It maybe that's why smokers are so underrepresented in the data. Their chronic hypoxia from smoking increases their hemoglobin counts. This also would also predict less severe acute mitochondrial failure from COVID 19 disease in high altitude populations. Nairobi is untouched.5. Hypoxia explains the loss of taste and smell too.human olfactory receptor becomes less sensitive under hypoxic hypoxia.
t. Dr Krauss
literally just take vitamin c and zinc
>healthitanalytics.com
"Higher levels of hemoglobin, the iron-containing protein that enables blood cells to carry oxygen to bodily tissues, were also strongly linked to subsequent respiratory distress. The team pointed out that this could be explained by other factors, such as unreported smoking of tobacco, which has been associated with increased hemoglobin levels."
I wonder about this part. Why would high hemoglobin be a risk factor if discoupled from smoking (you can take nicotine without smoking)
Thank you so much for your helpful input Sir, user's we need to distribute this.
Re smoking, I've no idea what to think about that:
>PHE and Dr Matt completely ignored a much larger survey, mind, which originated from Wuhan Zhongnan Hospital, in which medical staff were reportedly astonished to discover that of the patients admitted presenting with Covid-19, only 1.4 per cent were smokers (out of a population in which more than half of men smoke cigarettes). It looked very much as though smokers were far, far less likely to catch the illness in the first place. This tallies a little with another stat from that New England Journal of Medicine survey — only about 11 per cent of those admitted with the virus were current smokers, compared with about 27 per cent of the population at large who smoke (and a much larger proportion if you factor in age).
>Another Chinese study of 41 patients showed that of the 13 who were admitted to intensive care units, none were smokers. Just to muddy the waters a little further, a paper in the European Journal of Internal Medicine concluded that there was no greater significant risk to smokers from Covid-19 than to people who did not smoke.
spectator.co.uk
One of reads added, "It was noted in London, after the Great Plague of 1665, that none of the capital's tobacconists had died from the disease."
Found the kike
Is this even a real thread?
If the virus kills by inducing hemoglobin dysfunction, why would having MORE hemoglobin make the disease worse? That’s the opposite of what you’d expect.
>HCQ + Zinc would be really effective if it were attacking hemoglobin. HCQ alone would not.
Can you explain this? Or link to an explanation of the mechanism. Cheers
because free iron in the body is toxic and can accumulate on organs, causing damage
This goes contrary to the AI saying that high hemoglobin was a predictor for bad outcomes?
I understand that. However, if haemoglobin is being destroyed and releasing free iron ions, the oxygen SATURATION level (as detected by a sats monitor on your finger) will stay normal or near normal, as it detects the difference between oxygenated and deoxygenation Hb concentrations; destroyed Hb with no iron simply won’t show up at all.
Instead, a reduction in total oxygen carriage induced by this mechanism would show up as an acute Hb drop on serial blood tests. This doesn’t appear to be a feature of the disease anywhere
I guess you didn’t get the memo. Plaquenil is DEAD. It’s a failed Psyop.
