Asymptomatic viral infections; calling biochem fags

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Makes sense. Looks like some idiot mishandled a batch of viruses.

Or the chicoms seeded different strains in different places once theirs was beginning to take off.
Either they did this because it was an accident and they realized that it was better to have the whole world face this so that they aren't the only country that is economically devastated; or, they did it intentionally and had the weaker strain in first in China and then released the deadlier strian in other countries because they are tired of economic warfare and the western elites gave them the go ahead.

isn't that because it binds to specific proteins on cell wall that are expressed more when you have diabetes or some other illness? So if you are healthy/young infection is not that severe because virus enters your cells slower so your body have more time to deal with it and produces antibodies before onset of serious symptoms. That's just my unproven theory based on some random internet facts.

Difference races, different genetics and life styles. Could have a bunch of race-targeted medicine remedies if not MUH EQUALITY.

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>How the fuck does a virus range from randomly doing nothing to making swiss cheese of your lungs, all within a relatively similar population?

I could tell you but you won't believe me and I don't give a fuck anymore so I hope y'all die

I have done very little research into this hoax but I would buy the blood type hypothesis
The genes that determine certain blood cell antigens are the same for other cells in the body. Your blood type might affect likelihood of your cells absorbing the virus

No one knows the full complex answer but one thing to keep in mind is that your immune system is not just antibodies and T cells that is only the adaptive immune system which requires previous exposure or up to a week before doing any significant attack. You have another entire part of the immune system designed to attack both old and completely new threats immediately called the innate system. So part of the answer is it will depend on how well that system works. And that system will vary depending on both genetics and environment like everything else.

Niet paruskie it has nothing to do with that, this shit is a degenerate slayer

Just because all the those with severe reactions just so happened to have done 23andme or Ancestry DNA tests is just a coincidence.

It could differ depending on the startting point of the infection.

If the throad is infected the body has time to develop an answer before it can travel to the lung.
The other way around with lung infected first could be worse.

>Are their immune systems so efficient that the virus never reaches a critical mass of replication, all while not triggering a strong enough immune response (which usually are the symptoms)?
Yes, that is literally it.

>How can this vary so wildly between people humans?
Because people's immune systems significantly degrade in effectiveness as they age or develop chronic diseases. And since every old person these days is obese and walking around with diabeetus, COPD, heart disease etc. they're dropping like flies, while almost everyone under 50 is sub-clinical.

Pic related

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source: just trust me bro

This is not true.

A compelling argument.

It's about viral load assuming it's the same virus (how much virus a person has at a given time). The immune system could be lucky and had some reaction to this novel virus quickly enough such that it won't give off symptoms. Some people have better immune systems than others.

I'm totally convinced by this argument

First year medstudent here, I'm only in first year but we already start working on adaptative immunity.

I have some theories about this shit but we need to dig first we need to understand what viruses are for real, most of people think viruses are here for hurting us, or some species of bacteria, viruses are not alive they are juste adn in the air, it's juste a genetic code and when we catch it we are starting to mutate.
For example viruses are why we are no longer dropping eggs, some reptilian. Catch a virus 300 millions years ago, female start bloodung the hell out of their vagina reptoid but some survived and became the first mammalians.

This virus is certainly bio engined, I think he have a lot of serotypes much more than human blood for example human blood got 4 serotype
A, B, AB, O.
Imagine this virus got 40 serotypes, à bio engineered shit with 40 serotypes, once u catch it he first time ur adaptive immune protect u against Z type but if enter in contact with someone with another type like P type ur adaptative immune will send soldiers trained against Z serotypes and not P serotypes, then ur body is full cha he'd of useless anticorps and that's why they are usine Choloroquine like anglos in the Indians wars when they invented The GIN TONIC for cure Dengue

They have already decoded the RNA several times, always mostly the same RNA (a few mutations are to be expected). At least not enough to say there are different SARS viruses in circulation.

Only Tinfoil hats can deny this.

>How the fuck does a virus range from randomly doing nothing to making swiss cheese of your lungs, all within a relatively similar population?
Because your immune system fights it off. The only people who've been dying have had some sort of pre-existing medical complication or compromised immune systems.

Also some studies seems to show that O types people are more immune to this virus, it's seems logical because O type people are universal donator but can have any blood for any other expect O people, O type does not have antigènes on their surfaces it means theys does not have lock it's a paradox u can't hijack a door without a lock, I don't say that O are immune but they are more resistant to infection. (I didn't post link but u can verify everything)

because if its actually engineered, and its actually got a component of HIV in it, then it's not just a flu that's more infectious because it has an HIV component, its HIV that spreads like a flu.

this would also explain the "mystery illnesses" reported in the US late 2019. the coronavirus family was racing across the globe, bouncing around, each mutating, like a big multi-threaded algorithm searching for a viable combination of factors in order to multiply.

There are plenty of people not dying that still get their shit fucked up. I'm not talking about the 80 year olds here.

'Spreading while asymptomatic' doesn't mean you won't or will never get sick eventually, or even that you don't have it. Because the incubation time is long, the onset of the actual symptoms could be days or even weeks, but you might technically 'have' the disease from a physiological standpoint.

It's been discovered that you can transmit while you're asymptomatic with this new disease in particular. With many other common diseases, they usually require symptoms appear for it to spread. For example some variant of the common cold might require coughing of mucus to spread the virus, and clearly someone isn't asymptomatic if they are coughing and have a runny nose. Other diseases like genital warts may only spread when you actually have warts or lesions on your body, and then come in some sexual contact with someone, but if your warts are in a phase where they are dormant or clear up you generally aren't contagious despite still having the disease in your body.

With COVID-19, they discovered its possible to spread even without coughing, having a fever, spewing on people or touching them. This basically means that just regular breathing near someone while infected is enough to transfer it to them, furthermore it is more hardy that more common viruses and can stay on surfaces or infect certain bacteria for long periods of time. Since the incubation is long as I mentioned earlier, there's a possibility for you to spread it for days or weeks while being asymptomatic. It's possible to come in contact with the disease and not get infected at all, but this is probably very rare based on the mechanisms the disease uses to infect. It's more likely that people who have some type of immunity to COVID might have been infected by SARS or a similar virus before, and got immune from its mechanisms that way. Either that or just plain luck.

It's just the flu, bro. The flu kills almost a million worldwide every year and nobody gives a fuck, turn off the TV.

Race specific bioweapon

Now the race seems to be men (double victim rate).

The flu does this too. It is far from uniform in how it attacks tissues. I had a case in my childhood that went after muscle tissue and I was unable to walk for a couple of weeks. Each infection, even when dealing with a single genetic viral strain, is different and may or may not progress into major organ disruption. Immune responses differ, but so do viral attack vectors.

>that vast majority of people
>between 50 and 75%
>50%
>vast majority
opinion completely discarded

Cases here of the elderly, people that have had chemo/radiotherapy and have recovered from this.

An annoying cunt that makes vlogs has diabetes and has recovered from this. It's a strange one, the blood type hypothesis is a good one but I'm more inclined to believe that idiocy eclipses genius, meaning some retarded Xang Wu Fung Tofu Noodles went and dumped a bunch of different SARS samples in a back alley bin. Samples had their antigenic shift inside a horseshoe bat, then another animal before making that one giant leap for viralkind.

But who really knows, ARB medications should be trialled if they're not already.

It affects men more because of comorbidity. Men are more likely to smoke. Men are more likely to have cardiovascular disease. Men are more likely to be stressed and have high blood pressure. All those things make the symptoms of what COVID-19 does even worse, which is why their death rate is much higher. Their hearts and lungs simply give out.

Then you'd think India would be having high cases but they're not? Cramped slums, bacteria that are resistant to antibiotics (to the point they are engineering viruses to attack bacterial infections), smokers, high fat diet of ghee and cow urine.

Why aren't they dropping like flies?

Young healthy people are killed by a cytokine storm that is the result of antibody dependent enhancement, which itself is caused by subneutralizing antibodies. The severity of the disease seems to hinge on three things...

1. The ability of the host immune system to quickly ramp up and destroy the initial infection (mostly asymptomatic)...be young and healthy
2. The presence of existing subneutralizing antibodies that facilitate infection... Don't live in regions that are frequently exposed to similar coronaviruses
3. Susceptibility to cytokine storms... likely to be genetic, we have some target genes already, but it's not a well understood area of medical biology

Source... Microbiology PhD

Good doctors, shalom! I have not brought my medical credentials with me, my apologies! I thought I was headed to the political forum, not the laboratory. Excuse me!

Take your meds

idiot animal.
that cow urine is healthier and cleaner than your daily meals.

it avoids altering the dna of mixed race people.

is cow urine high in fat? asking for a friend

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this is why they are not reporting the races of the people dying. a lot of chinese are pure blood chinese. same with africans in africa (which will be DEVASTATED in the coming weeks)

>microbiology phd
wtf is cytokine and wtf is a cytokine storm

Strength of Immune system

Some people get sick a lot. Some people "never get sick."

Virus enters all the time, if youre immune system is strong enough it will fuck the virus up and you dont even notice it (nothingburger).

Immune systems break down over time (with age) and the state of your immune system can also change quickly, due to stress, lack of sleep, eating like a burger, etc.

The answer is not even the top researchers know yet OP. However if we take it step by step, maybe we can figure it out.

Recall that it binds to ACE2 (which as an aside, is why the anti-malarials are showing effectiveness - they affect the same receptors). More ACE2 = higher chance of being infected. Therefore in theory anyone with a higher number of these receptors has a higher chance of being infected or having more severe symptoms due to more receptors harboring the virus: Anyone on ACE inhibitors or ARBs are a prime group to consider

Another possibility is some people have an innate hypersensitivity to the virus and/or its effects. Kind of like how people with Selective IgA deficiency have a higher frequency and more severe reactions to blood transfusions. These immunodeficiencies are surprisingly common, and it wouldnt be completely unprecedented if the people with the worst reactions have a common immunodeficiency

You also have to consider societal factors. India was one of the first nations to start to respond early by closing/restricting their borders. The poor people with lower hygiene you speak of in high-density areas also don't really travel that much which lowers the incidence of external inoculations of the virus in their communities. India is also not a hotspot for Chinese travel, whereas most of the other places that first contracted the disease are. India's production of cheap medicine also rivals china, I have no idea what they are doing there but I wouldn't be surprised if they are feeding their symptomatic cases some of the other proven treatments like old malaria drugs down their throats like skittles. Also higher temperatures especially in the southern regions where the poverty and higher density is common, which could have a profound impact on how quickly the virus spreads.

Medfag here. We simply do not know enough about how the body actually works to make anything more than educated guesses about the exact mechanisms at play here. Personally I see it more as a God's Will sort of thing.

There's multiple strains that were released.

It is probably going to have something to do with the microbiome of the lung and/or gut-lung axis.

A few related studies:

ncbi.nlm.nih.gov/pubmed/32140452
ncbi.nlm.nih.gov/pubmed/32130898
ncbi.nlm.nih.gov/pubmed/32117259

Just think of it as a severe auto immune response like rheumatoid arthritis, except that in this case the inflammation is typically affecting the cardiovascular system

>60 posts in
>no plebbit doctor word soup copypasta
you've lost your touch Yas Forums

What is a cytokine storm?
A cytokine storm is an overproduction of immune cells and their activating compounds (cytokines), which, in a flu infection, is often associated with a surge of activated immune cells into the lungs.Feb 27, 2014

That's not true at all the Chinese have been subjugated rape mutts going back many dynasties.